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1 A Single F153Sβ3 Mutation Causes Constitutive Integrin αIIbβ3 Activation in a Variant Form of
This report identifies a novel variant form of the inherited bleeding disorder, Glanzmann thrombasthenia (GT) exhibiting only mild in
2 A Single F153Sβ3 Mutation Causes Constitutive Integrin αIIbβ3 Activation in a Variant Form of
This report identifies a novel variant form of the inherited bleeding disorder, Glanzmann thrombasthenia (GT) exhibiting only mild ble
3 A Single F153Sβ3 Mutation Causes Constitutive Integrin αIIbβ3 Activation in a Variant Form of
This report identifies a novel variant form of the inherited bleeding disorder, Glanzmann thrombasthenia (GT) exhibiting only mild ble
4 NCOG-34. A DESCRIPTIVE ANALYSIS OF GLIOMATOSIS CEREBRI CASES, COMPARED ACCORDING TO IDH STATUS
1 2 3 4 7 a (5 10 16 19 3+ 43 GC II IV Of On RT as at be by is no of on or to 3/7 6/7 AA, All GC, IDH Ten The age all and had our the
5 NCOG-34. A DESCRIPTIVE ANALYSIS OF GLIOMATOSIS CEREBRI CASES, COMPARED ACCORDING TO IDH STATUS
1 2 3 4 7 a (5 10 16 19 3+ 43 GC II IV Of On RT as at be by is no of on or to 3/7 6/7 AA, All GC, IDH Ten The age all and had our the
6 NCOG-39. EXPLORING PATIENT REPORTED OUTCOMES (PROS) ACROSS ETHNORACIAL GROUPS IN PRIMARY BRAIN T
( = a p (p 5% 50 6% AA be in is no of on or to 10% 28% 29% 40% 44% 562 79% AND KPS PBT The age all and but for had has may not per the
7 NCOG-39. EXPLORING PATIENT REPORTED OUTCOMES (PROS) ACROSS ETHNORACIAL GROUPS IN PRIMARY BRAIN T
( = a p (p 5% 50 6% AA be in is no of on or to 10% 28% 29% 40% 44% 562 79% AND KPS PBT The age all and but for had has may not per the

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