전문정보검색
| No | 제목 / 내용 |
|---|---|
| 1 |
Only the strong survive: The impact of frailty on patients undergoing leadless pacemaker implant Medical complexity, as defined by the number and weighting of comorbidities, is a significant contributor to morbidity, mortality risk |
| 2 |
Only the Strong Survive: The Impact of Frailty on Patients Undergoing Leadless Pacemaker Implant Medical complexity, as defined by the number and weighting of comorbidities, is a significant contributor to morbidity, mortality risk |
| 3 |
The Role of Endoplasmic Reticulum in Lipotoxicity during Metabolic Dysfunction–Associated Stea Perturbations in lipid and protein homeostasis induce endoplasmic reticulum (ER) stress metabolic dysfunction–associated steatotic l |
| 4 |
Evolving real-world patterns of practice in metastatic castration-sensitive prostate cancer (mCS 86 Background: Treatment options for patients with mCSPC have rapidly evolved the introduction of androgen receptor axis targeted ther |
| 5 |
Modeling foot sole cutaneous afferents: FootSim •FootSim simulates a realistic neural activation of foot sole afferents•This in silico tool is suggested for neuroprosthetic and n |
| 6 |
Obesity control by SHIP inhibition requires pan-paralog inhibition and an intact eosinophil comp •Pan-SHIP1/2 inhibitors (pan-SHIPi) reverse diet-induced obesity and hyperglycemia•Prevention of (DIO) by pan-SHIPi requires eosin |
| 7 |
Stress and Liver Fibrogenesis Stress response pathways are crucial for cells to adapt physiological and pathologic conditions. Increased transcription translation i |
| 8 |
Photovoltaics literature survey (No. 183) In order to help readers stay up-to-date in the field, each issue of Progress Photovoltaics will contain a list recently published jou |
| 9 |
Acknowledgment to Reviewers The Clinical Cancer Research editors wish to acknowledge with sincere appreciation the assistance of following reviewers who have gene |
| 10 |
ISRIB Blunts the Integrated Stress Response by Allosterically Antagonising the Inhibitory Effect •Mutually antagonistic binding of phosphorylated eIF2 and ISRIB to eIF2B•Opposing structural rearrangement eIF2B by or eIF2•Phos |